FNDC3B is another novel partner fused to RARA in the t(3;17)(q26;q21) variant of acute promyelocytic leukemia-Reference-Cited by-同舟云学术

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FNDC3B is another novel partner fused to RARA in the t(3;17)(q26;q21) variant of acute promyelocytic leukemia

Published:2017-05-11 Issue:19 Volume:129 Page:2705-2709
ISSN:0006-4971
Container-title:Blood
language:en
Short-container-title:

Author:

Cheng Chi Keung¹,Wang Angela Z.¹,Wong Terry H. Y.¹,Wan Thomas S. K.¹,Cheung Joyce S.¹,Raghupathy Radha²,Chan Natalie P. H.¹,Ng Margaret H. L.¹³

Affiliation:

1. Blood Cancer Cytogenetics and Genomics Laboratory, Department of Anatomical and Cellular Pathology, Prince of Wales Hospital,

2. Department of Clinical Oncology, Prince of Wales Hospital, and

3. State Key Laboratory in Oncology in South China, The Chinese University of Hong Kong, Hong Kong SAR, China

Publisher

American Society of Hematology

Subject

Cell Biology,Hematology,Immunology,Biochemistry

Link

http://ashpublications.org/blood/article-pdf/129/19/2705/1400610/blood767707.pdf

Reference15 articles.

1. TBLR1 fuses to retinoid acid receptor α in a variant t(3;17)(q26;q21) translocation of acute promyelocytic leukemia;Chen;Blood,2014

2. HDAC inhibitors lead to significant survival benefit in a novel fusion protein TBLR1-RARα induced murine promyelocytic leukemia model [abstract];Li;Blood,2016

3. The novel gene fad104, containing a fibronectin type III domain, has a significant role in adipogenesis;Tominaga;FEBS Lett,2004

4. Proteomics. Tissue-based map of the human proteome;Uhlén;Science,2015

5. Deconstructing a disease: RARalpha, its fusion partners, and their roles in the pathogenesis of acute promyelocytic leukemia [review];Melnick;Blood,1999

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4. Identifying STRN3-RARA as a new fusion gene for acute promyelocytic leukemia;Blood;2023-10-26

5. Clinical and genomic characterization of an ATRA‐insensitive acute promyelocytic leukemia variant with a FNDC3B::RARB fusion;Genes, Chromosomes and Cancer;2023-06-07

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