GATA4-dependent regulation of the secretory phenotype via MCP-1 underlies lamin A-mediated human mesenchymal stem cell aging
Author:
Publisher
Springer Science and Business Media LLC
Subject
Clinical Biochemistry,Molecular Biology,Molecular Medicine,Biochemistry
Link
http://www.nature.com/articles/s12276-018-0092-3.pdf
Reference41 articles.
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2. Scaffidi, P. & Misteli, T. Lamin A-dependent nuclear defects in human aging. Science312, 1059–1063 (2006).
3. Liu, B. et al. Genomic instability in laminopathy-based premature aging. Nat. Med.11, 780–785 (2005).
4. Liu, Y., Rusinol, A., Sinensky, M., Wang, Y. & Zou, Y. DNA damage responses in progeroid syndromes arise from defective maturation of prelamin A. J. Cell Sci.119(Pt 22), 4644–4649 (2006).
5. Pendás, A. M. et al. Defective prelamin A processing and muscular and adipocyte alterations in Zmpste24 metalloproteinase–deficient mice.Nat. Genet.31, 94–99 (2002).
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