Hepatic hydrogen sulfide levels are reduced in mouse model of Hutchinson-Gilford progeria syndrome

Author:

Wilkie Stephen E.12,Marcu Diana E.1,Carter Roderick N.3,Morton Nicholas M.3,Gonzalo Susana4,Selman Colin1

Affiliation:

1. Glasgow Ageing Research Network (GARNER), School of Biodiversity, One Health and Veterinary Medicine, College of Medical, Veterinary and Life Sciences, University of Glasgow, Glasgow G12 8QQ, UK

2. Division of Molecular Metabolism, Department of Medical Biochemistry and Biophysics, Karolinska Institute, Solna 171 65, Sweden

3. Molecular Metabolism Group, University/BHF Centre for Cardiovascular Sciences, Queens Medical Research Institute, University of Edinburgh, Edinburgh EH16 4TJ, UK

4. Department of Biochemistry and Molecular Biology, Edward A. Doisy Research Center, Saint Louis University School of Medicine, MO 63104, USA

Publisher

Impact Journals, LLC

Subject

Cell Biology,Aging

Reference48 articles.

1. Model of human aging: recent findings on Werner's and Hutchinson-Gilford progeria syndromes.;Shen;Clin Interv Aging,2008

2. Pathology of Mouse Models of Accelerated Aging.;de Bruin;Vet Pathol,2016

3. Hutchinson-Gilford progeria syndrome as a model for vascular aging.;Hoesli;Biogerontology,2016

4. Mouse models in modeling aging and cancer.;Luo;Exp Gerontol,2019

5. Questions, Gordon. The Progeria Research Foundation. 2006; 1-6. https://www.progeriaresearch.org/.

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