Benzbromarone Stabilizes ΔF508 CFTR at the Cell Surface
Author:
Affiliation:
1. Departments of Medicine and Biochemistry, University of Toronto, Toronto, Ontario M5S 1A8, Canada
Publisher
American Chemical Society (ACS)
Subject
Biochemistry
Link
https://pubs.acs.org/doi/pdf/10.1021/bi2004813
Reference32 articles.
1. Cystic Fibrosis
2. Identification of the Cystic Fibrosis Gene: Cloning and Characterization of Complementary DNA
3. Most F508del-CFTR Is Targeted to Degradation at an Early Folding Checkpoint and Independently of Calnexin
4. Defective intracellular transport and processing of CFTR is the molecular basis of most cystic fibrosis
5. The Cystic Fibrosis-causing Mutation ΔF508 Affects Multiple Steps in Cystic Fibrosis Transmembrane Conductance Regulator Biogenesis
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3. Stability of the ABCD1 Protein with a Missense Mutation: A Novel Approach to Finding Therapeutic Compounds for X-Linked Adrenoleukodystrophy;JIMD Reports;2018
4. ΔF508 CFTR Surface Stability Is Regulated by DAB2 and CHIP-Mediated Ubiquitination in Post-Endocytic Compartments;PLOS ONE;2015-04-16
5. Synthesis of heterocycle-tethered acylbenzofurans and benzodifurans from odorless and recyclable organoseleno polystyrene resin;RSC Adv.;2014
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