Defective intracellular transport and processing of CFTR is the molecular basis of most cystic fibrosis-Reference-Cited by-同舟云学术

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Defective intracellular transport and processing of CFTR is the molecular basis of most cystic fibrosis

Published:1990-11 Issue:4 Volume:63 Page:827-834
ISSN:0092-8674
Container-title:Cell
language:en
Short-container-title:Cell

Author:

Cheng Seng H.,Gregory Richard J.,Marshall John,Paul Sucharita,Souza David W.,White Gary A.,O'Riordan Catherine R.,Smith Alan E.

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference32 articles.

1. Cystic fibrosis;Boat,1989

2. Peptide antibodies to the human c-fyn gene product demonstrate pp59c-fyn is capable of complex formation with the middle-T antigen of polyomavirus;Cheng;EMBO J.,1988

3. Peptide mapping by limited proteolysis in sodium dodecyl sulfate and analysis by gel electrophoresis;Cleveland;J. Biol. Chem.,1977

4. Construction of biologically functional bacterial plasmids in vitro;Cohen,1973

5. A cluster of cystic fibrosis mutations in the first nucleotide binding fold of the cystic fibrosis conductance regulator protein;Cutting;Nature,1990

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