Polyamidoamine dendrimer-conjugated triamcinolone acetonide attenuates nerve injury-induced spinal cord microglia activation and mechanical allodynia-Reference-Cited by-同舟云学术

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Polyamidoamine dendrimer-conjugated triamcinolone acetonide attenuates nerve injury-induced spinal cord microglia activation and mechanical allodynia

Published:2017-01 Issue: Volume:13 Page:174480691769700
ISSN:1744-8069
Container-title:Molecular Pain
language:en
Short-container-title:Mol Pain

Author:

Kim Hwisung¹,Choi Boomin¹,Lim Hyoungsub¹,Min Hyunjung¹,Oh Jae Hoon²,Choi Sunghyun²,Cho Joung Goo³,Park Jong-Sang²,Lee Sung Joong¹

Affiliation:

1. Department of Neuroscience and Physiology, Dental Research Institute, School of Dentistry, Seoul National University, Seoul, Republic of Korea

2. School of Chemistry and Molecular Engineering, Seoul National University, Seoul, Republic of Korea

3. Department of Anesthesiology and Pain Medicine, National Health Insurance Service Ilsan Hospital, Ilsan, Republic of Korea

Publisher

SAGE Publications

Subject

Anesthesiology and Pain Medicine,Cellular and Molecular Neuroscience,Molecular Medicine

Link

http://journals.sagepub.com/doi/pdf/10.1177/1744806917697006

Reference41 articles.

1. GLIA: A novel drug discovery target for clinical pain

2. Spinal blockade of TNF blocks spinal nerve ligation-induced increases in spinal P-p38

3. Cytokine Mechanisms of Central Sensitization: Distinct and Overlapping Role of Interleukin-1 , Interleukin-6, and Tumor Necrosis Factor- in Regulating Synaptic and Neuronal Activity in the Superficial Spinal Cord

4. BDNF from microglia causes the shift in neuronal anion gradient underlying neuropathic pain

5. Identification and characterization of triamcinolone acetonide, a microglial-activation inhibitor

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