Cutting Edge: Oral Type I IFN-τ Promotes a Th2 Bias and Enhances Suppression of Autoimmune Encephalomyelitis by Oral Glatiramer Acetate-Reference-Cited by-同舟云学术

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Cutting Edge: Oral Type I IFN-τ Promotes a Th2 Bias and Enhances Suppression of Autoimmune Encephalomyelitis by Oral Glatiramer Acetate

Published:2002-09-01 Issue:5 Volume:169 Page:2231-2235
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Soos Jeanne M.,Stüve Olaf,Youssef Sawsan,Bravo Manuel,Johnson Howard M.,Weiner Howard L.,Zamvil Scott S.

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference23 articles.

1. Interferon beta in the treatment of multiple sclerosis

2. Mechanisms of action of glatiramer acetate in multiple sclerosis

3. Immunomodulatory effects of interferon beta-1a in multiple sclerosis

4. Interferon (IFN) beta acts downstream of IFN-gamma-induced class II transactivator messenger RNA accumulation to block major histocompatibility complex class II gene expression and requires the 48-kD DNA-binding protein, ISGF3-gamma.

5. Specific inhibition of the T-cell response to myelin basic protein by the synthetic copolymer Cop 1.

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2. Protective Effects of Interferon-tau Against Lipopolysaccharide-Induced Embryo Implantation Failure in Pregnant Mice;Journal of Interferon & Cytokine Research;2018-05

3. The Evolving Mechanisms of Action of Glatiramer Acetate;Cold Spring Harbor Perspectives in Medicine;2018-02-12

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