Intracellular retention of mutant lysyl oxidase leads to aortic dilation in response to increased hemodynamic stress
Author:
Funder
National Institutes of Health
Ines Mandl Research Foundation
Publisher
American Society for Clinical Investigation
Subject
General Medicine
Link
https://insight.jci.org/articles/view/127748/files/pdf
Reference46 articles.
1. Therapeutics Targeting Drivers of Thoracic Aortic Aneurysms and Acute Aortic Dissections: Insights from Predisposing Genes and Mouse Models
2. Variants of Unknown Significance in Genes Associated with Heritable Thoracic Aortic Disease Can Be Low Penetrant “Risk Variants”
3. Loss of function mutation in LOX causes thoracic aortic aneurysm and dissection in humans
4. Enzymatic and non-enzymatic functions of the lysyl oxidase family in bone
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