GSK-3β/mTORC1 Couples Synaptogenesis and Axonal Repair to Reduce Hypoxia Ischemia-Mediated Brain Injury in Neonatal Rats
Author:
Affiliation:
1. Department of Pediatrics, West China Second University Hospital and Key Laboratory of Obstetrics & Gynecologic and Pediatric Diseases and Birth Defects of the Ministry of Education, Sichuan University, Sichuan, China
Publisher
Oxford University Press (OUP)
Subject
Cellular and Molecular Neuroscience,Clinical Neurology,Neurology,General Medicine,Pathology and Forensic Medicine
Link
http://academic.oup.com/jnen/article-pdf/77/5/383/24615330/nly015.pdf
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3. Actualities on molecular pathogenesis and repairing processes of cerebral damage in perinatal hypoxic-ischemic encephalopathy;Distefano;Ital J Pediatr,2010
4. Involvement of the Akt/GSK-3beta/CRMP-2 pathway in axonal injury after hypoxic-ischemic brain damage in neonatal rat;Xiong;Neuroscience,2012
5. CaMKII phosphorylates collapsin response mediator protein 2 and modulates axonal damage during glutamate excitotoxicity;Hou;J Neurochem,2009
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