Bumetanide Attenuates Cognitive Deficits and Brain Damage in Rats Subjected to Hypoxia–Ischemia at Two Time Points of the Early Postnatal Period-Reference-Cited by-同舟云学术

Bumetanide Attenuates Cognitive Deficits and Brain Damage in Rats Subjected to Hypoxia–Ischemia at Two Time Points of the Early Postnatal Period

Published:2023-06-28 Issue:6 Volume:41 Page:526-545
ISSN:1029-8428
Container-title:Neurotoxicity Research
language:en
Short-container-title:Neurotox Res

Author:

Machado Diorlon Nunes,Durán-Carabali Luz Elena,Odorcyk Felipe Kawa,Carvalho Andrey Vinicios Soares,Martini Ana Paula Rodrigues,Schlemmer Livia Machado,de Mattos Marcel de Medeiros,Bernd Gabriel Pereira,Dalmaz Carla,Netto Carlos Alexandre

Funder

Conselho Nacional de Desenvolvimento Científico e Tecnológico

Coordenação de Aperfeiçoamento de Pessoal de Nível Superior

Publisher

Springer Science and Business Media LLC

Subject

Toxicology,General Neuroscience

Link

https://link.springer.com/content/pdf/10.1007/s12640-023-00654-3.pdf

Reference82 articles.

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4. Ben-Ari Y, Tseeb V, Raggozzino D, Khazipov R, Gaiarsa JL (1994) γ-aminobutyric acid (GABA): a fast excitatory transmitter which may regulate the development of hippocampal neurones in early postnatal life. Prog Brain Res 102:261–273. https://doi.org/10.1016/S0079-6123(08)60545-2

5. Bie B, Wu J, Lin F, Naguib M, Xu J (2022) Suppression of hippocampal GABAergic transmission impairs memory in rodent models of Alzheimer’s disease. Eur J Pharmacol 917:174771. https://doi.org/10.1016/j.ejphar.2022.174771

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