CaMKII phosphorylates collapsin response mediator protein 2 and modulates axonal damage during glutamate excitotoxicity-Reference-Cited by-同舟云学术

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CaMKII phosphorylates collapsin response mediator protein 2 and modulates axonal damage during glutamate excitotoxicity

Published:2009-11 Issue:3 Volume:111 Page:870-881
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:

Author:

Hou Sheng T.,Jiang Susan X.,Aylsworth Amy,Ferguson Graeme,Slinn Jacqueline,Hu Houwen,Leung Thomas,Kappler Joachim,Kaibuchi Kozo

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1471-4159.2009.06375.x/fullpdf

Reference43 articles.

1. Phosphorylation of collapsin response mediator protein-2 by Rho-kinase. Evidence for two separate signaling pathways for growth cone collapse;Arimura;J. Biol. Chem.,2000

2. Role of CRMP-2 in neuronal polarity;Arimura;J. Neurobiol.,2004

3. Phosphorylation by Rho kinase regulates CRMP-2 activity in growth cones;Arimura;Mol. Cell. Biol.,2005

4. Calpain product of WT-CRMP2 reduces the amount of surface NR2B NMDA receptor subunit;Bretin;J. Neurochem.,2006

5. Alpha2-chimaerin, cyclin-dependent Kinase 5/p35, and its target collapsin response mediator protein-2 are essential components in semaphorin 3A-induced growth-cone collapse;Brown;J. Neurosci.,2004

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