Localized changes to glycogen synthase kinase-3 and collapsin response mediator protein-2 in the Huntington's disease affected brain-Reference-Cited by-同舟云学术

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Localized changes to glycogen synthase kinase-3 and collapsin response mediator protein-2 in the Huntington's disease affected brain

Published:2014-03-14 Issue:15 Volume:23 Page:4051-4063
ISSN:0964-6906
Container-title:Human Molecular Genetics
language:en
Short-container-title:Human Molecular Genetics

Author:

Lim N. K. H.,Hung L. W.,Pang T. Y.,Mclean C. A.,Liddell J. R.,Hilton J. B.,Li Q.-X.,White A. R.,Hannan A. J.,Crouch P. J.

Publisher

Oxford University Press (OUP)

Subject

Genetics (clinical),Genetics,Molecular Biology,General Medicine

Reference55 articles.

1. Huntingtonʼs disease

2. A new model for prediction of the age of onset and penetrance for Huntington's disease based on CAG length

3. Evidence that glycogen synthase kinase-3 isoforms have distinct substrate preference in the brain

4. Glycogen Synthase Kinase-3: Properties, Functions, and Regulation

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1. Untangling the Role of Tau in Huntington’s Disease Pathology;Journal of Huntington's Disease;2023-05-02

2. GSK-3β Allosteric Inhibition: A Dead End or a New Pharmacological Frontier?;International Journal of Molecular Sciences;2023-04-19

3. Structure–activity relationship (SAR) studies on substituted N-(pyridin-3-yl)-2-amino-isonicotinamides as highly potent and selective glycogen synthase kinase-3 (GSK-3) inhibitors;Bioorganic & Medicinal Chemistry Letters;2023-02

4. Role of GSK-3β Inhibitors: New Promises and Opportunities for Alzheimer’s Disease;Advanced Pharmaceutical Bulletin;2023-01-23

5. A generic method for fluorescence monitoring glycogen through patent blue V triggered supramolecular switching;Sensors and Actuators B: Chemical;2022-05

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