Deficiency of Ube3a in Huntington's disease mice brain increases aggregate load and accelerates disease pathology-Reference-Cited by-同舟云学术

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Deficiency of Ube3a in Huntington's disease mice brain increases aggregate load and accelerates disease pathology

Published:2014-07-04 Issue:23 Volume:23 Page:6235-6245
ISSN:0964-6906
Container-title:Human Molecular Genetics
language:en
Short-container-title:

Author:

Maheshwari Megha,Shekhar Shashi,Singh Brijesh Kumar,Jamal Imran,Vatsa Naman,Kumar Vipendra,Sharma Ankit,Jana Nihar Ranjan

Publisher

Oxford University Press (OUP)

Subject

Genetics(clinical),Genetics,Molecular Biology,General Medicine

Reference62 articles.

1. Trinucleotide repeat disorders;Annu. Rev. Neurosci.,2007

2. Neuropathological diagnosis and CAG repeat expansion in Huntington's disease;J. Neurol. Neurosurg. Psychiatry.,1996

3. Diseases of unstable repeat expansion: mechanisms and common principles;Nat. Rev. Genet.,2005

4. A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes. The Huntington's Disease Collaborative Research Group;Cell,1993

5. Androgen receptor gene mutations in X-linked spinal and bulbar muscular atrophy;Nature,1991

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1. Ubiquitin-modifying enzymes in Huntington’s disease;Frontiers in Molecular Biosciences;2023-02-08

2. Huntingtin Ubiquitination Mechanisms and Novel Possible Therapies to Decrease the Toxic Effects of Mutated Huntingtin;Journal of Personalized Medicine;2021-12-06

3. Praja1 ubiquitin ligase facilitates degradation of polyglutamine proteins and suppresses polyglutamine-mediated toxicity;Molecular Biology of the Cell;2021-08-15

4. Withaferin A Induces Heat Shock Response and Ameliorates Disease Progression in a Mouse Model of Huntington’s Disease;Molecular Neurobiology;2021-04-26

5. Identification of UBE3A Protein in CSF and Extracellular Space of the Hippocampus Suggest a Potential Novel Function in Synaptic Plasticity;Autism Research;2021-01-20

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