A novel FGFR3-binding peptide inhibits FGFR3 signaling and reverses the lethal phenotype of mice mimicking human thanatophoric dysplasia-Reference-Cited by-同舟云学术

A novel FGFR3-binding peptide inhibits FGFR3 signaling and reverses the lethal phenotype of mice mimicking human thanatophoric dysplasia

Published:2012-09-26 Issue:26 Volume:21 Page:5443-5455
ISSN:1460-2083
Container-title:Human Molecular Genetics
language:en
Short-container-title:

Author:

Jin Min,Yu Ying,Qi Huabing,Xie Yangli,Su Nan,Wang Xiaofeng,Tan Qiaoyan,Luo Fengtao,Zhu Ying,Wang Quan,Du Xiaolan,Xian Cory J.,Liu Peng,Huang Haiyang,Shen Yue,Deng Chu-Xia,Chen Di,Chen Lin

Publisher

Oxford University Press (OUP)

Subject

Genetics (clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/article-pdf/21/26/5443/17257502/dds390.pdf

Reference59 articles.

1. Roles of epidermal growth factor family in the regulation of postnatal somatic growth;Xian;Endocr. Rev.,2007

2. Mutations in the transmembrane domain of FGFR3 cause the most common genetic form of dwarfism, achondroplasia;Shiang;Cell,1994

3. Fibroblast growth factor receptor 3 and the human chondrodysplasias;Horton;Curr. Opin. Pediatr.,1997

4. Common mutations in the fibroblast growth factor receptor 3 (FGFR 3) gene account for achondroplasia, hypochondroplasia, and thanatophoric dwarfism;Bonaventure;Am. J. Med. Genet.,1996

5. Missense FGFR3 mutations create cysteine residues in thanatophoric dwarfism type I (TD1);Rousseau;Hum. Mol. Genet.,1996

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