Severe acute respiratory syndrome Coronavirus ORF3a protein activates the NLRP3 inflammasome by promoting TRAF3‐dependent ubiquitination of ASC-Reference-Cited by-同舟云学术

Severe acute respiratory syndrome Coronavirus ORF3a protein activates the NLRP3 inflammasome by promoting TRAF3‐dependent ubiquitination of ASC

Published:2019-04-29 Issue:8 Volume:33 Page:8865-8877
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Siu Kam‐Leung¹,Yuen Kit‐San¹,Castano‐Rodriguez Carlos²,Ye Zi‐Wei³,Yeung Man‐Lung¹,Fung Sin‐Yee¹,Yuan Shuofeng³,Chan Chi‐Ping¹,Yuen Kwok‐Yung³,Enjuanes Luis²,Jin Dong‐Yan¹

Affiliation:

1. School of Biomedical Sciences The University of Hong Kong Pokfulam Hong Kong

2. Department of Molecular and Cell Biology Centro Nacional de Biotecnología‐Consejo Superior de Investigaciones Cientificas (CNB‐CSIC) Madrid Spain

3. Department of Microbiology The University of Hong Kong Pokfulam Hong Kong

Funder

Health and Medical Research Fund

National Institutes of Health

National Institute of Allergy and Infectious Diseases

European Commission

Innovative Medicines Initiative

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.201802418R

Reference69 articles.

1. Middle East Respiratory Syndrome Coronavirus: Another Zoonotic Betacoronavirus Causing SARS-Like Disease

2. Comparative Host Gene Transcription by Microarray Analysis Early after Infection of the Huh7 Cell Line by Severe Acute Respiratory Syndrome Coronavirus and Human Coronavirus 229E

3. Functional Genomics Highlights Differential Induction of Antiviral Pathways in the Lungs of SARS-CoV–Infected Macaques

4. Inflammasomes: mechanism of assembly, regulation and signalling

5. NALP3 Forms an IL-1β-Processing Inflammasome with Increased Activity in Muckle-Wells Autoinflammatory Disorder

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