GTP cyclohydrolase I gene transfer reverses tetrahydrobiopterin deficiency and increases nitric oxide synthesis in endothelial cells and isolated vessels from diabetic rats-Reference-Cited by-同舟云学术

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GTP cyclohydrolase I gene transfer reverses tetrahydrobiopterin deficiency and increases nitric oxide synthesis in endothelial cells and isolated vessels from diabetic rats

Published:2004-10-05 Issue:15 Volume:18 Page:1900-1902
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Meininger Cynthia J.¹,Cai Shijie²,Parker Janet L.¹,Channon Keith M.²,Kelly Katherine A.¹,Becker Elizabeth J.¹,Wood M. Kathy³,Wade Laura A.¹,Wu Guoyao¹⁴

Affiliation:

1. Cardiovascular Research Institute/Department of Medical PhysiologyTexas A&M University System Health Science CenterTempleTX76504

2. Department of Cardiovascular MedicineUniversity of Oxford, John Radcliffe HospitalOxfordUK

3. Department of BiologyUniversity of Mary Hardin‐BaylorBeltonTX76513

4. Department of Animal ScienceTexas A&M UniversityCollege StationTX77843

Funder

National Institutes of Health

American Heart Association

British Heart Foundation

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.04-1702fje

Reference25 articles.

1. Endothelial dysfunction in diabetes

2. Impaired arginine metabolism and NO synthesis in coronary endothelial cells of the spontaneously diabetic BB rat;Wu G.;Am. J. Physiol.,1995

3. Impaired endothelium-dependent vasodilation in patients with insulin-dependent diabetes mellitus.

4. Nitric oxide and vascular responses in Type I diabetes

5. Impaired nitric oxide production in coronary endothelial cells of the spontaneously diabetic BB rat is due to tetrahydrobiopterin deficiency

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4. Oxidative Stress, GTPCH1, and Endothelial Nitric Oxide Synthase Uncoupling in Hypertension;Antioxidants & Redox Signaling;2021-03-20

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