Inhibition of protein translation as a novel mechanism for prostaglandin E 2 regulation of cell functions-Reference-Cited by-同舟云学术

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Inhibition of protein translation as a novel mechanism for prostaglandin E 2 regulation of cell functions

Published:2013-09-26 Issue:1 Volume:28 Page:56-66
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Okunishi Katsuhide¹,DeGraaf Angela J.¹,Zasłona Zbigniew¹,Peters‐Golden Marc¹

Affiliation:

1. Division of Pulmonary and Critical Care MedicineDepartment of Internal MedicineUniversity of Michigan Health SystemAnn ArborMichiganUSA

Funder

Deutsche Forschungsgemeinschaft

National Institutes of Health

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.13-231720

Reference50 articles.

1. Common and unique mechanisms regulate fibrosis in various fibroproliferative diseases

2. Different Effects of Growth Factors on Proliferation and Matrix Production of Normal and Fibrotic Human Lung Fibroblasts

3. Human alveolar macrophage inhibition of lung fibroblast growth: a prostaglandin‐dependent process;Elias J. A.;Am. Rev. Respir. Dis.,1985

4. Modulation of alveolar macrophage-driven fibroblast proliferation by alternative macrophage mediators.

5. The differential effect of prostaglandin E2 on transforming growth factor‐beta and insulin‐induced collagen formation in lung fibroblasts;Fine A.;J. Biol. Chem.,1989

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