Prion Peptide PrP106-126 Induces Inducible Nitric Oxide Synthase and Proinflammatory Cytokine Gene Expression Through the Activation of NF-κB in Macrophage Cells-Reference-Cited by-同舟云学术

Prion Peptide PrP106-126 Induces Inducible Nitric Oxide Synthase and Proinflammatory Cytokine Gene Expression Through the Activation of NF-κB in Macrophage Cells

Published:2012-05 Issue:5 Volume:31 Page:833-838
ISSN:1044-5498
Container-title:DNA and Cell Biology
language:en
Short-container-title:DNA and Cell Biology

Author:

Lu Yun¹,Liu Ailing¹²,Zhou Xiangmei¹,Kouadir Mohammed¹,Zhao Wei¹,Zhang Siming¹,Yin Xiaomin¹,Yang Lifeng¹,Zhao Deming¹

Affiliation:

1. State Key Laboratories for Agrobiotechnology, Key Lab of Animal Epidemiology and Zoonosis, Ministry of Agriculture, National Animal Transmissible Spongiform Encephalopathy Laboratory, College of Veterinary Medicine, China Agricultural University, Beijing, China.

2. China Animal Health and Epidemiology Center, Qingdao, Shandong, China.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Genetics,Molecular Biology,General Medicine

Link

http://www.liebertpub.com/doi/pdf/10.1089/dna.2011.1362

Reference26 articles.

1. Amyloid -peptide stimulates nitric oxide production in astrocytes through an NF B-dependent mechanism

2. The cellular prion protein modulates phagocytosis and inflammatory response

3. Activation by prion peptide PrP 106–126 induces a NF‐κB‐driven proinflammatory response in human monocyte‐derived dendritic cells

4. The inducible transcription activator NF-κB: regulation by distinct protein subunits

5. NF-κB: Ten Years After

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