Activation by prion peptide PrP 106–126 induces a NF‐κB‐driven proinflammatory response in human monocyte‐derived dendritic cells-Reference-Cited by-同舟云学术

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Activation by prion peptide PrP 106–126 induces a NF‐κB‐driven proinflammatory response in human monocyte‐derived dendritic cells

Published:2003-05-22 Issue:1 Volume:74 Page:118-125
ISSN:0741-5400
Container-title:Journal of Leukocyte Biology
language:en
Short-container-title:Journal of Leukocyte Biology

Author:

Bacot Silvia M.¹,Lenz Petra²,Frazier‐Jessen Michelle R.¹,Feldman Gerald M.¹

Affiliation:

1. Division of Monoclonal Antibodies, Center for Biologics Evaluation and Research, Food and Drug Administration, Bethesda, Maryland; and

2. Laboratory of Cellular Oncology, National Cancer Institute, National Institutes of Health, Bethesda, Maryland

Funder

Oak Ridge Institute for Science and Education

National Institute of Arthritis and Musculoskeletal and Skin Diseases

National Institutes of Health

Publisher

Wiley

Subject

Cell Biology,Immunology,Immunology and Allergy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1189/jlb.1102521

Reference53 articles.

1. GENETICS OF PRIONS

2. Cellular isoform of the scrapie agent protein participates in lymphocyte activation

3. Prion protein is necessary for normal synaptic function

4. Evidence for the Conformation of the Pathologic Isoform of the Prion Protein Enciphering and Propagating Prion Diversity

5. Infected splenic dendritic cells are sufficient for prion transmission to the CNS in mouse scrapie

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