Pathogenic prion protein fragment (PrP106–126) promotes human immunodeficiency virus type-1 infection in peripheral blood monocyte-derived macrophages
Author:
Funder
U.S. Food and Drug Administration
National Institute of Dental and Craniofacial Research
Publisher
Elsevier BV
Subject
Virology
Reference25 articles.
1. Activation by prion peptide PrP106–126 induces a NF-κB-driven proinflammatory response in human monocyte-derived dendritic cells;Bacot;J. Leukoc. Biol.,2003
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3. Cellular isoform of the scrapie agent protein participates in lymphocyte activation;Cashman;Cell,1990
4. Hemin activation ameliorates HIV-1 infection via heme oxygenase-1 induction;Devadas;J. Immunol.,2006
5. The stimulation of inducible nitric synthase by the prion protein fragment 106–126 in human microglia is tumor necrosis factor-a-dependent and involves p38 mitogen-activated protein kinase;Fabrizi;J. Biol. Chem.,2001
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