Redox Control of Calcium Channels: From Mechanisms to Therapeutic Opportunities-Reference-Cited by-同舟云学术

Redox Control of Calcium Channels: From Mechanisms to Therapeutic Opportunities

Published:2007-04 Issue:4 Volume:9 Page:409-435
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Hool Livia C.¹,Corry Ben²

Affiliation:

1. Discipline of Physiology, School of Biomedical, Biomolecular, and Chemical Sciences, The University of Western Australia, Crawley, Western Australia.

2. Discipline of Chemistry, School of Biomedical, Biomolecular, and Chemical Sciences, The University of Western Australia, Crawley, Western Australia.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2006.1446

Reference299 articles.

1. Structure-Functional Diversity of Human L-Type Ca2+Channel: Perspectives for New Pharmacological Targets: Figure 1

2. Sulfhydryl oxidation and Ca2+ release from sarcoplasmic reticulum

3. S-Glutathiolation by peroxynitrite activates SERCA during arterial relaxation by nitric oxide

4. Nitric Oxide Protects the Skeletal Muscle Ca2+Release Channel from Oxidation Induced Activation

5. Mechanistically Distinct Steps in the Mitochondrial Death Pathway Triggered by Oxidative Stress in Cardiac Myocytes

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