Effect of Short Periods of Normobaric Hyperoxia on Local Brain Tissue Oxygenation and Cerebrospinal Fluid Oxidative Stress Markers in Severe Traumatic Brain Injury-Reference-Cited by-同舟云学术

Effect of Short Periods of Normobaric Hyperoxia on Local Brain Tissue Oxygenation and Cerebrospinal Fluid Oxidative Stress Markers in Severe Traumatic Brain Injury

Published:2009-08 Issue:8 Volume:26 Page:1241-1249
ISSN:0897-7151
Container-title:Journal of Neurotrauma
language:en
Short-container-title:Journal of Neurotrauma

Author:

Puccio Ava M.¹²,Hoffman Leslie A.³,Bayir Hülya⁴,Zullo Thomas G.³,Fischer Michael¹,Darby Joseph⁵,Alexander Sheila³,Dixon C. Edward¹²,Okonkwo David O.¹²,Kochanek Patrick M.⁴⁶

Affiliation:

1. Department of Neurological Surgery, University of Pittsburgh, Pittsburgh, Pennsylvania.

2. Brain Trauma Research Center, University of Pittsburgh, Pennsylvania.

3. Department of Acute/Tertiary Care, University of Pittsburgh School of Nursing, Pittsburgh, Pennsylvania.

4. Department of Pediatrics and Critical Care Medicine, University of Pittsburgh and Children's Hospital of Pittsburgh, Pennsylvania.

5. Department of Critical Care Medicine, University of Pittsburgh Medical Center, Pittsburgh, Pennsylvania.

6. Safar Center for Resuscitative Research, University of Pittsburgh, Pennsylvania.

Publisher

Mary Ann Liebert Inc

Subject

Neurology (clinical)

Link

http://www.liebertpub.com/doi/pdf/10.1089/neu.2008.0624

Reference18 articles.

1. Assessment of Antioxidant Reserves and Oxidative Stress in Cerebrospinal Fluid after Severe Traumatic Brain Injury in Infants and Children

2. Postischemic inhibition of cerebral cortex pyruvate dehydrogenase

3. Effect of hyperoxia on cerebral metabolic rate for oxygen measured using positron emission tomography in patients with acute severe head injury

4. Inflicted Childhood Neurotrauma: New Insight into The Detection, Pathobiology, Prevention, and Treatment of Our Youngest Patients with Traumatic Brain Injury

5. Brain tissue oxygen tension response to induced hyperoxia reduced in hypoperfused brain

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