Enhanced Store Overload–Induced Ca 2+ Release and Channel Sensitivity to Luminal Ca 2+ Activation Are Common Defects of RyR2 Mutations Linked to Ventricular Tachycardia and Sudden Death-Reference-Cited by-同舟云学术

Enhanced Store Overload–Induced Ca 2+ Release and Channel Sensitivity to Luminal Ca 2+ Activation Are Common Defects of RyR2 Mutations Linked to Ventricular Tachycardia and Sudden Death

Published:2005-11-25 Issue:11 Volume:97 Page:1173-1181
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circulation Research

Author:

Jiang Dawei¹,Wang Ruiwu¹,Xiao Bailong¹,Kong Huihui¹,Hunt Donald J.¹,Choi Philip¹,Zhang Lin¹,Chen S. R. Wayne¹

Affiliation:

1. From the Cardiovascular Research Group, Departments of Physiology and Biophysics, and of Biochemistry and Molecular Biology, University of Calgary, Alberta, Canada.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference33 articles.

1. Electrophysiological changes in heart failure and their relationship to arrhythmogenesis

2. Cellular Basis of Triggered Arrhythmias in Heart Failure

3. Genes, exercise and sudden death: molecular basis of familial catecholaminergic polymorphic ventricular tachycardia

4. Clinical and Molecular Characterization of Patients With Catecholaminergic Polymorphic Ventricular Tachycardia

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