Can cardiomyocytes bypass the ‘Ca2+-induced Ca2+-release’ mechanism?
Author:
Affiliation:
1. Inserm, UMR-S 1180, ‘Signaling and Cardiovascular Pathophysiology’, Université Paris-Saclay , 17 Avenue des Sciences , 91400 Orsay, France
Funder
National Agency for Research
Europe H2020
National Institute of Health
CSC
Publisher
Oxford University Press (OUP)
Subject
Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology
Link
https://academic.oup.com/cardiovascres/advance-article-pdf/doi/10.1093/cvr/cvad193/55674647/cvad193.pdf
Reference8 articles.
1. Calcium-induced release of calcium from the cardiac sarcoplasmic reticulum;Fabiato;Am J Physiol,1983
2. Mutations in the cardiac ryanodine receptor gene (hRyR2) underlie catecholaminergic polymorphic ventricular tachycardia;Priori;Circulation,2001
3. Enhanced store overload-induced Ca2+ release and channel sensitivity to luminal Ca2+ activation are common defects of RyR2 mutations linked to ventricular tachycardia and sudden death;Jiang;Circ Res,2005
4. Impaired binding to junctophilin-2 and nanostructural alteration in CPVT mutation;Yin;Circ Res,2021
5. Point mutations in RyR2 Ca2+ binding residues of human cardiomyocytes cause cellular remodelling of cardiac;Xia;Cardiovasc Res,2023
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