The Intracellular Cyclophilin PpiB Contributes to the Virulence of Staphylococcus aureus Independently of Its Peptidyl-Prolyl cis/trans Isomerase Activity
Author:
Affiliation:
1. Department of Biological Sciences, Ohio University, Athens, Ohio, USA
Abstract
Funder
NIAID
Publisher
American Society for Microbiology
Subject
Infectious Diseases,Immunology,Microbiology,Parasitology
Link
https://journals.asm.org/doi/pdf/10.1128/IAI.00379-18
Reference46 articles.
1. Lu T, DeLeo FR. 2015. Pathogenesis of Staphylococcus aureus in humans, p 711–748. In Singh SK (ed), Human emerging and re-emerging infections. John Wiley & Sons, Inc, Hoboken, NJ.
2. Increased Neutrophil Extracellular Trap–Mediated Staphylococcus aureus Clearance Through Inhibition of Nuclease Activity by Clindamycin and Immunoglobulin
3. Staphylococcus aureus Nuclease Is an SaeRS-Dependent Virulence Factor
4. Staphylococcus aureus Degrades Neutrophil Extracellular Traps to Promote Immune Cell Death
5. Temporal and Stochastic Control of Staphylococcus aureus Biofilm Development
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