Can't live with or without it: calcium and its role in Duchenne muscular dystrophy-induced muscle weakness. Focus on “SERCA1 overexpression minimizes skeletal muscle damage in dystrophic mouse models”
Author:
Affiliation:
1. Department of Physiology and Pharmacology, Karolinska Institute, Stockholm, Sweden; and
2. Department of Medicine, Solna, Cardiology Section, Karolinska University Hospital, Stockholm, Sweden
Publisher
American Physiological Society
Subject
Cell Biology,Physiology
Link
https://www.physiology.org/doi/pdf/10.1152/ajpcell.00056.2015
Reference9 articles.
1. Leaky ryanodine receptors in β-sarcoglycan deficient mice: a potential common defect in muscular dystrophy
2. Sarcolipin is a newly identified regulator of muscle-based thermogenesis in mammals
3. Increased fatigue resistance linked to Ca2+-stimulated mitochondrial biogenesis in muscle fibres of cold-acclimated mice
4. Mitigation of muscular dystrophy in mice by SERCA overexpression in skeletal muscle
5. Dystrophin: The protein product of the duchenne muscular dystrophy locus
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