Mitigation of muscular dystrophy in mice by SERCA overexpression in skeletal muscle-Reference-Cited by-同舟云学术

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Mitigation of muscular dystrophy in mice by SERCA overexpression in skeletal muscle

Published:2011-03-01 Issue:3 Volume:121 Page:1044-1052
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Goonasekera Sanjeewa A.,Lam Chi K.,Millay Douglas P.,Sargent Michelle A.,Hajjar Roger J.,Kranias Evangelia G.,Molkentin Jeffery D.

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference48 articles.

1. Emerging strategies for cell and gene therapy of the muscular dystrophies

2. Gene table of monogenic neuromuscular disorders (nuclear genome only)

3. Calcium and the damage pathways in muscular dystrophyThis article is one of a selection of papers published in this special issue on Calcium Signaling.

4. Increased Activity of Calcium Leak Channels in Myotubes of Duchenne Human and mdx Mouse Origin

5. The alteration of calcium homeostasis in adult dystrophic mdx muscle fibers is worsened by a chronic exercise in vivo

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1. Duchenne muscular dystrophy skeletal muscle cells derived from human induced pluripotent stem cells recapitulate various calcium dysregulation pathways;Cell Calcium;2024-11

2. SERCA2a overexpression improves muscle function in a canine Duchenne muscular dystrophy model;Molecular Therapy - Methods & Clinical Development;2024-06

3. GDE5/Gpcpd1 activity determines phosphatidylcholine composition in skeletal muscle and regulates contractile force in mice;Communications Biology;2024-05-20

4. MCU-independent Ca2+ uptake mediates mitochondrial Ca2+ overload and necrotic cell death in a mouse model of Duchenne muscular dystrophy;Scientific Reports;2024-03-21

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