Platelets from WAS patients show an increased susceptibility toex vivophagocytosis
Author:
Publisher
Informa UK Limited
Subject
Hematology,General Medicine
Link
http://www.tandfonline.com/doi/pdf/10.3109/09537104.2012.693991
Reference22 articles.
1. X-linked thrombocytopenia (XLT) due to WAS mutations: clinical characteristics, long-term outcome, and treatment options
2. Wiskott-Aldrich Syndrome at the nexus of autoimmune and primary immunodeficiency diseases
3. WASP is involved in proliferation and differentiation of human haemopoietic progenitorsin vitro
4. Deficiency in the Wiskott-Aldrich protein induces premature proplatelet formation and platelet production in the bone marrow compartment
5. X-linked thrombocytopenia caused by a mutation in the Wiskott-Aldrich syndrome (WAS) gene that disrupts interaction with the WAS protein (WASP)-interacting protein (WIP)
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