Anti-inflammatory effects of cinnamon extract and identification of active compounds influencing the TLR2 and TLR4 signaling pathways-Reference-Cited by-同舟云学术

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Anti-inflammatory effects of cinnamon extract and identification of active compounds influencing the TLR2 and TLR4 signaling pathways

Published:2018 Issue:11 Volume:9 Page:5950-5964
ISSN:2042-6496
Container-title:Food & Function
language:en
Short-container-title:Food Funct.

Author:

Schink Anne¹²³⁴,Naumoska Katerina¹²³⁴⁵^ORCID,Kitanovski Zoran¹²³⁴,Kampf Christopher Johannes¹²³⁴⁶^ORCID,Fröhlich-Nowoisky Janine¹²³⁴^ORCID,Thines Eckhard⁷⁸⁴⁹¹⁰,Pöschl Ulrich¹²³⁴^ORCID,Schuppan Detlef¹¹¹²¹³⁴¹⁴,Lucas Kurt¹²³⁴^ORCID

Affiliation:

1. Multiphase Chemistry Department

2. Max Planck Institute for Chemistry

3. 55128 Mainz

4. Germany

5. Department of Food Chemistry

6. Institute of Organic Chemistry

7. Institut für Biotechnologie und Wirkstoff Forschung gGmbH

8. Kaiserslautern

9. Institute of Molecular Physiology

10. Johannes Gutenberg University Mainz

11. Institute of Translational Immunology

12. University Medical Center of the Johannes Gutenberg University

13. 55131 Mainz

14. Division of Gastroenterology

Abstract

Cinnamon extract and its active compounds attenuate TLR2-/TLR4-mediated inflammatory processes.

Funder

Johannes Gutenberg-Universität Mainz

Javna Agencija za Raziskovalno Dejavnost RS

Deutscher Akademischer Austauschdienst

Publisher

Royal Society of Chemistry (RSC)

Subject

General Medicine,Food Science

Link

http://pubs.rsc.org/en/content/articlepdf/2018/FO/C8FO01286E

Reference65 articles.

1. Role of the Toll Like Receptor (TLR) Radical Cycle in Chronic Inflammation: Possible Treatments Targeting the TLR4 Pathway

2. Pattern recognition receptors—Molecular orchestrators of inflammation in inflammatory bowel disease

3. Akt-mediated regulation of NFκB and the essentialness of NFκB for the oncogenicity of PI3K and Akt

4. The emerging role of Toll-like receptor 4 in myocardial inflammation

5. A Large Subgroup of Mild-to-Moderate Asthma Is Persistently Noneosinophilic

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