Tenascin-C drives persistence of organ fibrosis-Reference-Cited by-同舟云学术

Tenascin-C drives persistence of organ fibrosis

Published:2016-06-03 Issue:1 Volume:7 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Bhattacharyya Swati,Wang Wenxia,Morales-Nebreda Luisa,Feng Gang,Wu Minghua,Zhou Xiaodong,Lafyatis Robert,Lee Jungwha,Hinchcliff Monique,Feghali-Bostwick Carol,Lakota Katja,Budinger G. R. Scott,Raparia Kirtee,Tamaki Zenshiro,Varga John

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/ncomms11703.pdf

Reference65 articles.

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3. Johnson, M. E. et al. Experimentally-derived fibroblast gene signatures identify molecular pathways associated with distinct subsets of systemic sclerosis patients in three independent cohorts. PloS One 10, e0114017 (2015).

4. Johnson, M. E., Pioli, P. A. & Whitfield, M. L. Gene expression profiling offers insights into the role of innate immune signaling in SSc. Semin. Immunopathol. 37, 501–509 (2015).

5. Piccinini, A. M. & Midwood, K. S. DAMPening inflammation by modulating TLR signalling. Mediators Inflamm. 2010, 1–21 (2010).

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