Loss of MUTYH function in human cells leads to accumulation of oxidative damage and genetic instability-Reference-Cited by-同舟云学术

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Loss of MUTYH function in human cells leads to accumulation of oxidative damage and genetic instability

Published:2012-10-29 Issue:38 Volume:32 Page:4500-4508
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Ruggieri V,Pin E,Russo M T,Barone F,Degan P,Sanchez M,Quaia M,Minoprio A,Turco E,Mazzei F,Viel A,Bignami M

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/onc2012479.pdf

Reference41 articles.

1. David SS, O’Shea VL, Kundu S . Base-excision repair of oxidative DNA damage. Nature 2007; 447: 941–950.

2. van Loon B, Markkanen E, Hübscher U . Oxygen as a friend and enemy: How to combat the mutational potential of 8-oxo-guanine. DNA Repair (Amst) 2010; 9: 604–616.

3. Oka S, Nakabeppu Y . DNA glycosylase encoded by MUTYH functions as a molecular switch for programmed cell death under oxidative stress to suppress tumorigenesis. Cancer Sci 2011; 102: 677–682.

4. Slupska MM, Luther WM, Chiang JH, Yang H, Miller JH . Functional expression of hMYH, a human homolog of the Escherichia coli MutY protein. J Bacteriol 1999; 181: 6210–6213.

5. Al-Tassan N, Chmiel NH, Maynard J, Fleming N, Livingston AL, Williams GT et al. Inherited variants of MYH associated with somatic G:C-->T:A mutations in colorectal tumors. Nat Genet 2002; 30: 227–232.

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2. Oxidative stress accelerates intestinal tumorigenesis by enhancing 8-oxoguanine-mediated mutagenesis in MUTYH-deficient mice;Genome Research;2024-01

3. Ergebnisse der endoskopischen Vorsorge und Therapie des Duodenums bei MUTYH-assoziierter Polyposis;Zentralblatt für Chirurgie - Zeitschrift für Allgemeine, Viszeral-, Thorax- und Gefäßchirurgie;2023-11-23

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