IL-10-producing intestinal macrophages prevent excessive antibacterial innate immunity by limiting IL-23 synthesis-Reference-Cited by-同舟云学术

IL-10-producing intestinal macrophages prevent excessive antibacterial innate immunity by limiting IL-23 synthesis

Published:2015-05-11 Issue:1 Volume:6 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Krause Petra,Morris Venetia,Greenbaum Jason A.,Park Yoon,Bjoerheden Unni,Mikulski Zbigniew^ORCID,Muffley Tracy,Shui Jr-Wen,Kim Gisen,Cheroutre Hilde,Liu Yun-Cai,Peters Bjoern^ORCID,Kronenberg Mitchell,Murai Masako^ORCID

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/ncomms8055.pdf

Reference49 articles.

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2. Moran, C. J. et al. IL-10R polymorphisms are associated with very-early-onset ulcerative colitis. Inflamm. Bowel. Dis. 19, 115–123 (2013).

3. Kuhn, R., Lohler, J., Rennick, D., Rajewsky, K. & Muller, W. Interleukin-10- deficient mice develop chronic enterocolitis. Cell 75, 263–274 (1993).

4. Spencer, S. D. et al. The orphan receptor CRF2-4 is an essential subunit of the interleukin 10 receptor. J. Exp. Med. 187, 571–578 (1998).

5. Yen, D. et al. IL-23 is essential for T cell-mediated colitis and promotes inflammation via IL-17 and IL-6. J. Clin. Invest. 116, 1310–1316 (2006).

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