A calcium sensor in the sodium channel modulates cardiac excitability-Reference-Cited by-同舟云学术

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A calcium sensor in the sodium channel modulates cardiac excitability

Published:2002-01 Issue:6870 Volume:415 Page:442-447
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Tan Hanno L.,Kupershmidt Sabina,Zhang Rong,Stepanovic Svetlana,Roden Dan M.,Wilde Arthur A. M.,Anderson Mark E.,Balser Jeffrey R.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/415442a.pdf

Reference30 articles.

1. Zuhlke, R. D. & Reuter, H. Ca2+-sensitive inactivation of L-type Ca2+ channels depends on multiple cytoplasmic amino acid sequences of the alpha1C subunit. Proc. Natl Acad. Sci. USA 95, 3287–3294 (1998).

2. Veldkamp, M. W. et al. Two distinct congenital arrhythmias evoked by a multidysfunctional Na+ channel. Circ. Res. 86, E91–E97 (2000).

3. Wang, D. W., Makita, N., Kitabatake, A., Balser, J. R. & George, A. L. Enhanced sodium channel intermediate inactivation in Brugada syndrome. Circ. Res. 87, E37–E43 (2000).

4. Chen, Q. et al. Genetic basis and molecular mechanism for idiopathic ventricular fibrillation. Nature 392, 293–296 (1998).

5. Dumaine, R. et al. Ionic mechanisms responsible for the electrocardiographic phenotype of the Brugada syndrome are temperature dependent. Circ. Res. 85, 803–809 (1999).

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