Mitochondrial PIP3-binding protein FUNDC2 supports platelet survival via AKT signaling pathway-Reference-Cited by-同舟云学术

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Mitochondrial PIP3-binding protein FUNDC2 supports platelet survival via AKT signaling pathway

Published:2018-05-21 Issue:2 Volume:26 Page:321-331
ISSN:1350-9047
Container-title:Cell Death & Differentiation
language:en
Short-container-title:Cell Death Differ

Author:

Ma Qi,Zhu Chongzhuo,Zhang Weilin,Ta Na,Zhang Rong,Liu Lei,Feng Du,Cheng Heping,Liu Junling,Chen Quan

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology

Link

http://www.nature.com/articles/s41418-018-0121-8.pdf

Reference42 articles.

1. Holmsen H. Physiological functions of platelets. Ann Med. 1989;21:23–30.

2. Lee SH, Du J, Stitham J, Atteya G, Lee S, Xiang Y, et al. Inducing mitophagy in diabetic platelets protects against severe oxidative stress. EMBO Mol Med. 2016;8:779–95.

3. Tyagi T, Ahmad S, Gupta N, Sahu A, Ahmad Y, Nair V, et al. Altered expression of platelet proteins and calpain activity mediate hypoxia-induced prothrombotic phenotype. Blood. 2014;123:1250–60.

4. McDonald TP, Cottrell M, Clift R. Effects of short-term hypoxia on platelet counts of mice. Blood. 1978;51:165–75.

5. Delaney MK, Liu J, Kim K, Shen B, Stojanovic-Terpo A, Zheng Y, et al. Agonist-induced platelet procoagulant activity requires shear and a Rac1-dependent signaling mechanism. Blood. 2014;124:1957–67.

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