A high incidence of WT1 abnormality in bilateral Wilms tumours in Japan, and the penetrance rates in children with WT1 germline mutation-Reference-Cited by-同舟云学术

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A high incidence of WT1 abnormality in bilateral Wilms tumours in Japan, and the penetrance rates in children with WT1 germline mutation

Published:2015-02-17 Issue:6 Volume:112 Page:1121-1133
ISSN:0007-0920
Container-title:British Journal of Cancer
language:en
Short-container-title:Br J Cancer

Author:

Kaneko Y,Okita H,Haruta M,Arai Y,Oue T,Tanaka Y,Horie H,Hinotsu S,Koshinaga T,Yoneda A,Ohtsuka Y,Taguchi T,Fukuzawa M

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Oncology

Link

http://www.nature.com/articles/bjc201513.pdf

Reference49 articles.

1. Cerrato F, Sparago A, Verde G, De Crescenzo A, Citro V, Cubellis MV, Rinaldi MM, Boccuto L, Neri G, Magnani C, D'Angelo P, Collini P, Perotti D, Sebastio G, Maher ER, Riccio A (2008) Different mechanisms cause imprinting defects at the IGF2/H19 locus in Beckwith-Wiedemann syndrome and Wilms' tumour. Hum Mol Genet 17 (10): 1427–1435.

2. Crow JF (2000) The origins, patterns and implications of human spontaneous mutation. Nat Rev Genet 1 (1): 40–47.

3. D'Angio GJ, Breslow N, Beckwith JB, Evans A, Baum H, deLorimier A, Fernbach D, Hrabovsky E, Jones B, Kelalis P, Othersen HB, Tefft M, Thomas PRM (1989) Treatment of Wilms' tumor. Results of the third national Wilms' tumor Study. Cancer 64 (2): 349–360.

4. Diller L, Ghahremani M, Morgan J, Grundy P, Reeves C, Breslow N, Green D, Neuberg D, Pelletier J, Li FP (1998) Constitutional WT1 mutations in Wilms' tumor patients. J Clin Oncol 16 (11): 3634–3640.

5. Dryja TP, Mukai S, Petersen R, Rapaport JM, Walton D, Yandell DW (1989) Parental origin of mutations of the retinoblastoma gene. Nature 339 (6225): 556–558.

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