Hyperglycaemia induces metabolic reprogramming into a glycolytic phenotype and promotes epithelial-mesenchymal transitions via YAP/TAZ-Hedgehog signalling axis in pancreatic cancer-Reference-Cited by-同舟云学术

Hyperglycaemia induces metabolic reprogramming into a glycolytic phenotype and promotes epithelial-mesenchymal transitions via YAP/TAZ-Hedgehog signalling axis in pancreatic cancer

Published:2022-12-19 Issue:5 Volume:128 Page:844-856
ISSN:0007-0920
Container-title:British Journal of Cancer
language:en
Short-container-title:Br J Cancer

Author:

Liu Zhao^ORCID,Hayashi Hiromitsu,Matsumura Kazuki,Ogata Yoko,Sato Hiroki,Shiraishi Yuta,Uemura Norio,Miyata Tatsunori,Higashi Takaaki,Nakagawa Shigeki,Mima Kosuke,Imai Katsunori,Baba Hideo^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Oncology

Link

https://www.nature.com/articles/s41416-022-02106-9.pdf

Reference47 articles.

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4. Norris AL, Roberts NJ, Jones S, Wheelan SJ, Papadoupoulous N, Vogelstein B, et al. Familial and sporadic pancreatic cancer share the same molecular pathogenesis. Fam Cancer. 2015;14:95–103.

5. Li S, Zhu H, Chen H, Xia J, Zhang F, Xu R, et al. Glucose promotes epithelial-mesenchymal transitions in bladder cancer by regulating the functions of YAP1 and TAZ. J Cell Mol Med. 2020;24:10391–401.

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