Pancreatic islets communicate with lymphoid tissues via exocytosis of insulin peptides-Reference-Cited by-同舟云学术

Pancreatic islets communicate with lymphoid tissues via exocytosis of insulin peptides

Published:2018-07-18 Issue:7716 Volume:560 Page:107-111
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Wan Xiaoxiao,Zinselmeyer Bernd H.,Zakharov Pavel N.,Vomund Anthony N.,Taniguchi Ruth,Santambrogio Laura,Anderson Mark S.,Lichti Cheryl F.,Unanue Emil R.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41586-018-0341-6.pdf

Reference32 articles.

1. Nakayama, M. et al. Prime role for an insulin epitope in the development of type 1 diabetes in NOD mice. Nature 435, 220–223 (2005).

2. Unanue, E. R. Antigen presentation in the autoimmune diabetes of the NOD mouse. Annu. Rev. Immunol. 32, 579–608 (2014).

3. Mohan, J. F. et al. Unique autoreactive T cells recognize insulin peptides generated within the islets of Langerhans in autoimmune diabetes. Nat. Immunol. 11, 350–354 (2010).

4. Mohan, J. F., Petzold, S. J. & Unanue, E. R. Register shifting of an insulin peptide–MHC complex allows diabetogenic T cells to escape thymic deletion. J. Exp. Med. 208, 2375–2383 (2011).

5. Mohan, J. F., Calderon, B., Anderson, M. S. & Unanue, E. R. Pathogenic CD4+ T cells recognizing an unstable peptide of insulin are directly recruited into islets bypassing local lymph nodes. J. Exp. Med. 210, 2403–2414 (2013).

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