Prime role for an insulin epitope in the development of type 1 diabetes in NOD mice-Reference-Cited by-同舟云学术

Prime role for an insulin epitope in the development of type 1 diabetes in NOD mice

Published:2005-05 Issue:7039 Volume:435 Page:220-223
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Nakayama Maki,Abiru Norio,Moriyama Hiroaki,Babaya Naru,Liu Edwin,Miao Dongmei,Yu Liping,Wegmann Dale R.,Hutton John C.,Elliott John F.,Eisenbarth George S.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/nature03523.pdf

Reference28 articles.

1. Wegmann, D. R., Norbury-Glaser, M. & Daniel, D. Insulin-specific T cells are a predominant component of islet infiltrates in pre-diabetic NOD mice. Eur. J. Immunol. 24, 1853–1857 (1994)

2. Lieberman, S. M. et al. Identification of the β cell antigen targeted by a prevalent population of pathogenic CD8+ T cells in autoimmune diabetes. Proc. Natl Acad. Sci. USA 100, 8384–8388 (2003)

3. Abiru, N. et al. Dual overlapping peptides recognized by insulin peptide B:9–23 T cell receptor AV13S3 T cell clones of the NOD mouse. J. Autoimmun. 14, 231–237 (2000)

4. Thébault-Baumont, K. et al. Acceleration of type 1 diabetes mellitus in proinsulin 2-deficient NOD mice. J. Clin. Invest. 111, 851–857 (2003)

5. Moriyama, H. et al. Evidence for a primary islet autoantigen (preproinsulin 1) for insulitis and diabetes in the nonobese diabetic mouse. Proc. Natl Acad. Sci. USA 100, 10376–10381 (2003)

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