Loss of lenalidomide-induced megakaryocytic differentiation leads to therapy resistance in del(5q) myelodysplastic syndrome-Reference-Cited by-同舟云学术

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Loss of lenalidomide-induced megakaryocytic differentiation leads to therapy resistance in del(5q) myelodysplastic syndrome

Published:2020-04-06 Issue:5 Volume:22 Page:526-533
ISSN:1465-7392
Container-title:Nature Cell Biology
language:en
Short-container-title:Nat Cell Biol

Author:

Martinez-Høyer Sergio,Deng Yu,Parker Jeremy^ORCID,Jiang Jihong,Mo Angela,Docking T. Roderick^ORCID,Gharaee Nadia,Li Jenny,Umlandt Patricia,Fuller Megan^ORCID,Jädersten Martin,Kulasekararaj Austin,Malcovati Luca^ORCID,List Alan F.,Hellström-Lindberg Eva,Platzbecker Uwe,Karsan Aly^ORCID

Funder

Associazione Italiana per la Ricerca sul Cancro

Leukemia and Lymphoma Society of Canada

Terry Fox Research Institute

Gouvernement du Canada | Canadian Institutes of Health Research

Genome British Columbia

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology

Link

http://www.nature.com/articles/s41556-020-0497-9.pdf

Reference36 articles.

1. Bejar, R., Levine, R. & Ebert, B. L. Unraveling the molecular pathophysiology of myelodysplastic syndromes. J. Clin. Oncol. 29, 504–515 (2011).

2. Gohring, G. et al. Patients with del(5q) MDS who fail to achieve sustained erythroid or cytogenetic remission after treatment with lenalidomide have an increased risk for clonal evolution and AML progression. Ann. Hematol. 89, 365–374 (2010).

3. Talati, C., Sallman, D. & List, A. F. SOHO state of the art and next questions: management of myelodysplastic syndromes with deletion 5q. Clin. Lymphoma Myeloma Leuk. 18, 629–635 (2018).

4. Kronke, J. et al. Lenalidomide induces ubiquitination and degradation of CK1α in del(5q) MDS. Nature 523, 183–188 (2015).

5. Fang, J. et al. A calcium- and calpain-dependent pathway determines the response to lenalidomide in myelodysplastic syndromes. Nat. Med. 22, 727–734 (2016).

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