The proline rich domain of p53 is dispensable for MGMT-dependent DNA repair and cell survival following alkylation damage-Reference-Cited by-同舟云学术

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The proline rich domain of p53 is dispensable for MGMT-dependent DNA repair and cell survival following alkylation damage

Published:2017-07-28 Issue:11 Volume:24 Page:1925-1936
ISSN:1350-9047
Container-title:Cell Death & Differentiation
language:en
Short-container-title:Cell Death Differ

Author:

Baran Katherine,Yang Mao,Dillon Christopher P,Samson Leona L,Green Douglas R

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology

Link

http://www.nature.com/articles/cdd2017116.pdf

Reference64 articles.

1. Lane DP . Cancer. p53, guardian of the genome. Nature 1992; 358: 15–16.

2. Bieging KT, Mello SS, Attardi LD . Unravelling mechanisms of p53-mediated tumour suppression. Nat Rev Cancer 2014; 14: 359–370.

3. Harvey M, McArthur MJ, Montgomery CA Jr., Butel JS, Bradley A, Donehower LA . Spontaneous and carcinogen-induced tumorigenesis in p53-deficient mice. Nat Genet 1993; 5: 225–229.

4. Donehower LA, Harvey M, Slagle BL, McArthur MJ, Montgomery CA Jr., Butel JS et al. Mice deficient for p53 are developmentally normal but susceptible to spontaneous tumours. Nature 1992; 356: 215–221.

5. Jacks T, Remington L, Williams BO, Schmitt EM, Halachmi S, Bronson RT et al. Tumor spectrum analysis in p53-mutant mice. Curr Biol 1994; 4: 1–7.

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