Uracil-DNA glycosylase is not implicated in the choice of the DNA repair pathway during B-cell class switch recombination
Author:
Publisher
Springer Science and Business Media LLC
Subject
Infectious Diseases,Immunology,Immunology and Allergy
Link
http://www.nature.com/articles/s41423-018-0034-y.pdf
Reference10 articles.
1. Chaudhuri, J. & Alt, F. W. Class-switch recombination: interplay of transcription, DNA deamination and DNA repair. Nat. Rev. Immunol. 4, 541–552 (2004).
2. Panchakshari, R. A. et al. DNA double-strand break response factors influence end-joining features of IgH class switch and general translocation junctions. Proc. Natl Acad. Sci. USA 115, 762–767 (2018).
3. Saintamand, A. et al. Elucidation of IgH 3’ region regulatory role during class switch recombination via germline deletion. Nat. Commun. 6, 7084 (2015).
4. Yousif, A. S., Stanlie, A., Mondal, S., Honjo, T. & Begum, N. A. Differential regulation of S-region hypermutation and class-switch recombination by noncanonical functions of uracil DNA glycosylase. Proc. Natl Acad. Sci. USA 111, E1016–E1024 (2014).
5. Yousif, A. S., Stanlie, A., Begum, N. A. & Honjo, T. Opinion: uracil DNA glycosylase (UNG) plays distinct and non-canonical roles in somatic hypermutation and class switch recombination. Int. Immunol. 26, 575–578 (2014).
Cited by 3 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Deletion of the 3′RR cis-enhancer IgH element affects DNA repair junctions during B-cell class switch recombination.;Immunology Letters;2021-12
2. Molecular analysis of γ1, γ3, and α class switch recombination junctions in APOBEC3-deficient mice using high-throughput sequencing;Cellular & Molecular Immunology;2019-10-08
3. Class switch recombination junctions are not affected by the absence of the immunoglobulin heavy chain Eμ enhancer;Cellular & Molecular Immunology;2019-04-09
1.学者识别学者识别
2.学术分析学术分析
3.人才评估人才评估
"同舟云学术"是以全球学者为主线，采集、加工和组织学术论文而形成的新型学术文献查询和分析系统，可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容，当前同舟云学术共收录了国内外主流学术期刊6万余种，收集的期刊论文及会议论文总量共计约1.5亿篇，并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询！咨询电话：010-8811{复制后删除}0370
www.globalauthorid.com
TOP
Copyright © 2019-2024 北京同舟云网络信息技术有限公司 京公网安备11010802033243号 京ICP备18003416号-3