Differential regulation of S-region hypermutation and class-switch recombination by noncanonical functions of uracil DNA glycosylase
Author:
Publisher
Proceedings of the National Academy of Sciences
Subject
Multidisciplinary
Reference57 articles.
1. Class Switch Recombination and Hypermutation Require Activation-Induced Cytidine Deaminase (AID), a Potential RNA Editing Enzyme
2. Discovery of Activation‐Induced Cytidine Deaminase, the Engraver of Antibody Memory
3. Activation-Induced Cytidine Deaminase-Dependent DNA Breaks in Class Switch Recombination Occur during G1 Phase of the Cell Cycle and Depend upon Mismatch Repair
4. The C-terminal region of activation-induced cytidine deaminase is responsible for a recombination function other than DNA cleavage in class switch recombination
5. Separate domains of AID are required for somatic hypermutation and class-switch recombination
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1. Molecular Mechanism of Activation-Induced Cytidine Deaminase;Molecular Biology of B Cells;2024
2. Necessity of HuR/ELAVL1 for the activation-induced cytidine deaminase-dependent decrease in topoisomerase 1 in antibody diversification;International Immunology;2023-04-22
3. Alternative end-joining in BCR gene rearrangements and translocations;Acta Biochimica et Biophysica Sinica;2022-05-01
4. Fam72a enforces error-prone DNA repair during antibody diversification;Nature;2021-11-24
5. SAMHD 1‐mediated dNTP degradation is required for efficient DNA repair during antibody class switch recombination;The EMBO Journal;2020-06-08
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