The miR-17∼92 family regulates the response to Toll-like receptor 9 triggering of CLL cells with unmutated IGHV genes-Reference-Cited by-同舟云学术

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The miR-17∼92 family regulates the response to Toll-like receptor 9 triggering of CLL cells with unmutated IGHV genes

Published:2012-02-20 Issue:7 Volume:26 Page:1584-1593
ISSN:0887-6924
Container-title:Leukemia
language:en
Short-container-title:Leukemia

Author:

Bomben R,Gobessi S,Dal Bo M,Volinia S,Marconi D,Tissino E,Benedetti D,Zucchetto A,Rossi D,Gaidano G,Del Poeta G,Laurenti L,Efremov D G,Gattei V

Publisher

Springer Science and Business Media LLC

Subject

Oncology,Cancer Research,Hematology

Link

http://www.nature.com/articles/leu201244.pdf

Reference60 articles.

1. Chiorazzi N, Rai KR, Ferrarini M . Chronic lymphocytic leukemia. N Engl J Med 2005; 352: 804–815.

2. Borche L, Lim A, Binet JL, Dighiero G . Evidence that chronic lymphocytic leukemia B lymphocytes are frequently committed to production of natural autoantibodies. Blood 1990; 76: 562–569.

3. Sthoeger ZM, Wakai M, Tse DB, Vinciguerra VP, Allen SL, Budman DR et al. Production of autoantibodies by CD5-expressing B lymphocytes from patients with chronic lymphocytic leukemia. J Exp Med 1989; 169: 255–268.

4. Coscia M, Pantaleoni F, Riganti C, Vitale C, Rigoni M, Peola S et al. IGHV unmutated CLL B cells are more prone to spontaneous apoptosis and subject to environmental prosurvival signals than mutated CLL B cells. Leukemia 2011; 25: 828–837.

5. Palacios F, Moreno P, Morande P, Abreu C, Correa A, Porro V et al. High expression of AID and active class switch recombination might account for a more aggressive disease in unmutated CLL patients: link with an activated microenvironment in CLL disease. Blood 2010; 115: 4488–4496.

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