Contribution of increased ISG15, ISGylation and deregulated type I IFN signaling in Usp18 mutant mice during the course of bacterial infections-Reference-Cited by-同舟云学术

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Contribution of increased ISG15, ISGylation and deregulated type I IFN signaling in Usp18 mutant mice during the course of bacterial infections

Published:2014-05-08 Issue:5 Volume:15 Page:282-292
ISSN:1466-4879
Container-title:Genes & Immunity
language:en
Short-container-title:Genes Immun

Author:

Dauphinee S M,Richer E,Eva M M,McIntosh F,Paquet M,Dangoor D,Burkart C,Zhang D-E,Gruenheid S,Gros P,Behr M,Malo D

Publisher

Springer Science and Business Media LLC

Subject

Genetics (clinical),Genetics,Immunology

Link

http://www.nature.com/articles/gene201417.pdf

Reference56 articles.

1. Crump JA, Mintz ED . Global trends in typhoid and paratyphoid fever. Clin Infect Dis 2010; 50: 241–246.

2. Vidal SM, Malo D, Marquis JF, Gros P . Forward genetic dissection of immunity to infection in the mouse. Annu Rev Immunol 2008; 26: 81–132.

3. Richer E, Prendergast C, Zhang DE, Qureshi ST, Vidal SM, Malo D . N-ethyl-N-nitrosourea-induced mutation in ubiquitin-specific peptidase 18 causes hyperactivation of IFN-alphabeta signaling and suppresses STAT4-induced IFN-gamma production, resulting in increased susceptibility to Salmonella typhimurium. J Immunol 2010; 185: 3593–3601.

4. Malakhova OA, Kim KI, Luo JK, Zou W, Kumar KG, Fuchs SY et al. UBP43 is a novel regulator of interferon signaling independent of its ISG15 isopeptidase activity. EMBO J 2006; 25: 2358–2367.

5. Kim KI, Yan M, Malakhova O, Luo JK, Shen MF, Zou W et al. Ube1L and protein ISGylation are not essential for alpha/beta interferon signaling. Mol Cell Biol 2006; 26: 472–479.

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