Somatic gene mutations expose cytoplasmic DNA to co-opt the cGAS/STING/NLRP3 axis in myelodysplastic syndromes
Author:
Funder
National cancer institute
Leukemia and Lymphoma Society
Publisher
American Society for Clinical Investigation
Subject
General Medicine
Link
https://insight.jci.org/articles/view/159430/files/pdf
Reference47 articles.
1. Induction of myelodysplasia by myeloid-derived suppressor cells
2. Toll-like Receptor-4 Is Up-Regulated in Hematopoietic Progenitor Cells and Contributes to Increased Apoptosis in Myelodysplastic Syndromes
3. The NLRP3 inflammasome functions as a driver of the myelodysplastic syndrome phenotype
4. TET2-mediated 5-hydroxymethylcytosine induces genetic instability and mutagenesis
5. Splicing Regulator SC35 Is Essential for Genomic Stability and Cell Proliferation during Mammalian Organogenesis
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