Overgrowth of a mouse model of Simpson– Golabi–Behmel syndrome is partly mediated by Indian Hedgehog
Author:
Affiliation:
1. Division of Molecular and Cell Biology, Sunnybrook Research Institute and Department of Medical Biophysics, University of Toronto 2075 Bayview Avenue Toronto Ontario M4N3M5 Canada
Publisher
EMBO
Subject
Genetics,Molecular Biology,Biochemistry
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1038/embor.2009.98
Reference21 articles.
1. Glypican-3–Deficient Mice Exhibit Developmental Overgrowth and Some of the Abnormalities Typical of Simpson-Golabi-Behmel Syndrome
2. Glypican-3 Inhibits Hedgehog Signaling during Development by Competing with Patched for Hedgehog Binding
3. Overgrowth of a Mouse Model of the Simpson–Golabi–Behmel Syndrome Is Independent of IGF Signaling
4. The glypican Dally-like is required for Hedgehog signalling in the embryonic epidermis of Drosophila
5. Hedgehog signaling in skeletal development
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