Overgrowth of a Mouse Model of the Simpson–Golabi–Behmel Syndrome Is Independent of IGF Signaling-Reference-Cited by-同舟云学术

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Overgrowth of a Mouse Model of the Simpson–Golabi–Behmel Syndrome Is Independent of IGF Signaling

Published:2002-03 Issue:1 Volume:243 Page:185-206
ISSN:0012-1606
Container-title:Developmental Biology
language:en
Short-container-title:Developmental Biology

Author:

Chiao Eric,Fisher Peter,Crisponi Laura,Deiana Manila,Dragatsis Ioannis,Schlessinger David,Pilia Giuseppe,Efstratiadis Argiris

Publisher

Elsevier BV

Subject

Cell Biology,Developmental Biology,Molecular Biology

Reference130 articles.

1. Transient dwarfism and hypogonadism in mice lacking Otx1 reveal prepubescent stage-specific control of pituitary levels of GH, FSH and LH;Acampora;Development,1998

2. Rescue of the limb deformity in hammertoe mutant mice by retinoic acid-induced cell death;Ahuja;Dev. Dyn.,1997

3. Zic1 regulates the patterning of vertebral arches in cooperation with Gli3;Aruga;Mech. Dev.,1999

4. Loss of Hoxa5 gene function in mice perturbs intestinal maturation;Aubin;Am. J. Physiol.,1999

5. Role of insulin-like growth factors in embryonic and postnatal growth;Baker;Cell,1993

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