β-catenin mediates glandular formation and dysregulation of β-catenin induces hyperplasia formation in the murine uterus-Reference-Cited by-同舟云学术

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β-catenin mediates glandular formation and dysregulation of β-catenin induces hyperplasia formation in the murine uterus

Published:2008-09-22 Issue:1 Volume:28 Page:31-40
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Jeong J-W,Lee H S,Franco H L,Broaddus R R,Taketo M M,Tsai S Y,Lydon J P,DeMayo F J

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/onc2008363.pdf

Reference64 articles.

1. Aberle H, Bauer A, Stappert J, Kispert A, Kemler R . (1997). Beta-catenin is a target for the ubiquitin-proteasome pathway. Embo J 16: 3797–3804.

2. Anderson MC, Robby SJ, Russell P . (2002). Endometritis, metaplasia, polyp and miscellaneous changes. Churchill Livingstone: London, 285–303 pp.

3. Arango NA, Szotek PP, Manganaro TF, Oliva E, Donahoe PK, Teixeira J . (2005). Conditional deletion of beta-catenin in the mesenchyme of the developing mouse uterus results in a switch to adipogenesis in the myometrium. Dev Biol 288: 276–283.

4. Brault V, Moore R, Kutsch S, Ishibashi M, Rowitch DH, McMahon AP et al. (2001). Inactivation of the beta-catenin gene by Wnt1-Cre-mediated deletion results in dramatic brain malformation and failure of craniofacial development. Development 128: 1253–1264.

5. Buchanan DL, Setiawan T, Lubahn DB, Taylor JA, Kurita T, Cunha GR et al. (1999). Tissue compartment-specific estrogen receptor-alpha participation in the mouse uterine epithelial secretory response. Endocrinology 140: 484–491.

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