Single and Transient Ca2+ Peaks in Podocytes do not induce Changes in Glomerular Filtration and Perfusion-Reference-Cited by-同舟云学术

Single and Transient Ca2+ Peaks in Podocytes do not induce Changes in Glomerular Filtration and Perfusion

Published:2016-10-19 Issue:1 Volume:6 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Koehler Sybille,Brähler Sebastian,Kuczkowski Alexander,Binz Julia,Hackl Matthias J.,Hagmann Henning,Höhne Martin,Vogt Merly C.,Wunderlich Claudia M.,Wunderlich F. Thomas,Schweda Frank,Schermer Bernhard,Benzing Thomas,Brinkkoetter Paul T.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep35400.pdf

Reference42 articles.

1. Pavenstädt, H., Kriz, W. & Kretzler, M. Cell biology of the glomerular podocyte. Physiol. Rev. 83, 253–307 (2003).

2. Benzing, T. Signaling at the Slit Diaphragm. J. Am. Soc. Nephrol. 15, 1382–1391 (2004).

3. Garg, P., Verma, R. & Holzman, L. B. Slit diaphragm junctional complex and regulation of the cytoskeleton. Nephron Exp. Nephrol. 106, e67–e72 (2007).

4. Huber, T. B. & Benzing, T. The slit diaphragm: a signaling platform to regulate podocyte function. Curr. Opin. Nephrol. Hypertens. 14, 211–216 (2005).

5. Boute, N. et al. NPHS2, encoding the glomerular protein podocin, is mutated in autosomal recessive steroid-resistant nephrotic syndrome. Nat. Genet. 24, 349–354 (2000).

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