Conserved S/T Residues of the Human Chaperone DNAJB6 Are Required for Effective Inhibition of Aβ42 Amyloid Fibril Formation-Reference-Cited by-同舟云学术

Conserved S/T Residues of the Human Chaperone DNAJB6 Are Required for Effective Inhibition of Aβ42 Amyloid Fibril Formation

Published:2018-07-19 Issue:32 Volume:57 Page:4891-4902
ISSN:0006-2960
Container-title:Biochemistry
language:en
Short-container-title:Biochemistry

Author:

Månsson Cecilia¹,van Cruchten Remco T. P.¹,Weininger Ulrich²,Yang Xiaoting¹,Cukalevski Risto¹,Arosio Paolo³^ORCID,Dobson Christopher M.³,Knowles Tuomas³⁴^ORCID,Akke Mikael²^ORCID,Linse Sara¹^ORCID,Emanuelsson Cecilia¹^ORCID

Affiliation:

1. Department of Biochemistry and Structural Biology, Center for Molecular Protein Science, Lund University, 221 00 Lund, Sweden

2. Department of Biophysical Chemistry, Center for Molecular Protein Science, Lund University, 221 00 Lund, Sweden

3. Department of Chemistry and Center for Misfolding Diseases, University of Cambridge, Cambridge CB2 1EW, U.K.

4. Cavendish Laboratory, Department of Physics, JJ Thomson Avenue, Cambridge CB3 0HE, U.K.

Funder

Vetenskapsr?det

FP7 People: Marie-Curie Actions

G?ran Gustafssons Stiftelse f?r Naturvetenskaplig och Medicinsk Forskning

European Commission

Magnus Bergvalls Stiftelse

Knut och Alice Wallenbergs Stiftelse

Wellcome Trust

China Scholarship Council

FP7 Ideas: European Research Council

Frances and Augustus Newman Foundation

Forskarskolan i L?kemedelsvetenskap, Lunds Universitet

Centre for Misfolding Diseases, University of Cambridge

NanoLund

Publisher

American Chemical Society (ACS)

Subject

Biochemistry

Link

https://pubs.acs.org/doi/pdf/10.1021/acs.biochem.8b00353

Reference65 articles.

1. Protein Misfolding, Amyloid Formation, and Human Disease: A Summary of Progress Over the Last Decade

2. The Amyloid Hypothesis of Alzheimer's Disease: Progress and Problems on the Road to Therapeutics

3. Protein aggregation diseases: pathogenicity and therapeutic perspectives

4. The amyloid hypothesis of Alzheimer's disease at 25 years

5. Alzheimer's Disease Mechanisms and Emerging Roads to Novel Therapeutics

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